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Issue title: Behavioral Neurology
Guest editors: David S. GeldmacherGuest Editor
Article type: Research Article
Authors: Kertesz, Andrew
Affiliations: Department of Clinical Neurological Sciences, St. Joseph's Health Centre, University of Western Ontario, London, Ontario N6A 4V2, Canada | Case Western Reserve University, Cleveland, Ohio, USA
Abstract: Recovery from aphasia is presented as a model of compensation after vascular or traumatic brain damage. Initial severity, time from onset, and etiology are the major prognostic factors. Initial severity is closely related to the size and location of the lesion. Asphasic syndromes reflect the deficit in various language networks. Lesion studies suggest that ipsilateral connected cortex plays a major role in compensation; contralateral contribution may occur in large lesions. Articulated language output network includes Broca's area, rolandic operculum, anterior insula, and the striatum. The comprehension network includes the superior posterior temporal gyrus and temporal operculum, the supramarginal gyrus and the angular gyrus.
Keywords: Recovery, Aphasia, Articulated language, Comprehension, Network model
DOI: 10.3233/NRE-1995-5202
Journal: NeuroRehabilitation, vol. 5, no. 2, pp. 103-113, 1995
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